yeah but that was probably a study done by a funded lab, be interesting to see what actual vapers say…although then again…knowing the meaning behind it, the numbers still probably wouldn’t be accurate, worth a shot tho, just for interest purposes.
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Considering smoking causes a myriad of lung related problems, I wouldn’t even want the numbers from smokers skewing the results. In fact, I would question the intent of any such study, because it would seem to be an attempt to make the number of cases higher, then lump vapers into those ststistics.
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For instance, consider the landscape of e-cigarette use in China, the first epicenter of COVID-19. A 2018 survey of 10 233 individuals in five Chinese cities found that only 0.9% had used e-cigarettes within the past 30 days [8]. Only 0.2% of those 65 years and older reported e-cigarette use within the past 30 days compared to 1.5% of those in the 15–24 year age range. Similarly, in 2016, of 32 931 adults included in the United States National Health Interview Survey, 1.0% of those over 65 years reported current e-cigarette use compared to 4.6% of the 18–44 year age group [9]. Older age groups, the ones more likely to have severe COVID-19, present to a hospital, and therefore be captured by epidemiologists in their surveys, are therefore less likely to report current vaping. On the other hand, it may be difficult to ascertain the prevalence of vaping in younger age groups who are much more likely to vape, but also much more likely to have mild or asymptomatic COVID-19 infections that are not captured either for their failure to present to a health care provider or the constraints placed on available tests in resource-limited settings. Nonetheless, we would argue for hospitals to capture these data as best they can and hope that data for mild cases in younger outpatients begin to be reported from around the world. Similar to smoking, it is possible that vaping may still be associated with worse outcomes, if not necessarily being a risk factor for contracting infection in the first place.
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Very early on I looked at how the coronavirus “worked” and there was a lot of talk about a specific enzyme found in the lungs - ACE2
ACE2 is more prevelant in smokers
At first there was talk that an abundance of ACE2 made it more difficult for the coronavirus to attach, then I read that not having a lot of it made it more difficult for the coronavirus, maybe somewhere inbetween is a “sweet spot” for the coronavirus.
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Beat me to it! 
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In regards to PG
A 2018 human volunteer study found that 10 male and female subjects undergoing 4 hours exposures to concentrations of up to 442 mg/m3 and 30 minutes exposures to concentrations of up to 871 mg/m3 in combination with moderate exercise did not show pulmonary function deficits, or signs of ocular irritation, with only slight symptoms of respiratory irritation reported
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Fascinating stuff guys…
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